Fibromyalgie-Patienten hören oft die Worte „Du siehst so gut aus. Wie können Sie krank sein? “Oder vielleicht die unsensiblen Worte” Fibromyalgie ist alles in Ihrem Kopf “. Laufende und aktuelle Forschung; bestätigt jedoch die Realität dieser unsichtbaren Krankheit. Diese Informationen ermöglichen es Personen mit Fibromyalgie und anderen ähnlichen unsichtbaren Krankheiten, zweifelsfrei voranzukommen und eine Bestätigung für ihre komplexen Krankheiten zu finden. Noch wichtiger ist, dass das Verständnis der jüngsten Forschung dabei hilft, die Richtung vorzugeben und natürliche Entscheidungen für eine gesunde Schmerztherapie mit Fibromyalgie zu identifizieren.
Was ist eine Entzündung?
Entzündung ist Teil des Heilungsprozesses des Körpers. Ohne Entzündung würden Infektionen und Wunden nicht heilen. Entzündungen können jedoch auch potenziell schädlich sein. Es gibt zwei Arten von Entzündungen, akute und chronische. Akute Entzündungen treten plötzlich aufgrund einer Verletzung oder Infektion auf. Es zeigt klassische Symptome wie Schwellung, Rötung und Schmerzen.
Die akute Entzündung ist vorübergehend und dauert je nach Entzündungsursache einige Tage bis einige Wochen. Chronische Entzündung ist eine langfristige Entzündung, die Monate und Jahre anhält. Es entsteht langsam und bereitet die Bühne für chronische Krankheiten. Herzerkrankungen, Autoimmunerkrankungen, neurologische Erkrankungen, Diabetes, Krebs, Alzheimer, Arthritis und viele andere Erkrankungen sind mit chronischen Entzündungen verbunden.
Forscherteams finden weit verbreitete Entzündungen im Gehirn von Fibromyalgie-Patienten
PET-Bildgebungsstudien zeigen eine erhöhte Gliaaktivierung, die mit dem Ermüdungsgrad korreliert. Eine Studie hat zum ersten Mal eine weit verbreitete Entzündung im Gehirn von Patienten mit der als Fibromyalgie bezeichneten, schlecht verstandenen Erkrankung dokumentiert.
Eine Studie von Forschern des Massachusetts General Hospital (MGH), die mit einem Team des Karolinska Institutet in Schweden zusammengearbeitet haben, hat zum ersten Mal eine weit verbreitete Entzündung im Gehirn von Patienten mit der schlecht verstandenen Erkrankung namens Fibromyalgie dokumentiert. Ihr Bericht wurde online in der Fachzeitschrift Brain, Behavior and Immunity veröffentlicht .
„Wir haben keine guten Behandlungsoptionen für Fibromyalgie, daher könnte die Identifizierung eines potenziellen Behandlungsziels zur Entwicklung innovativer, wirksamerer Therapien führen“, sagt Dr. Marco Loggia vom Martinos Center for Biomedical Imaging, MGH. leitender Autor des Berichts. “Und objektive neurochemische Veränderungen im Gehirn von Patienten mit Fibromyalgie sollten dazu beitragen, das anhaltende Stigma, dem viele Patienten ausgesetzt sind, zu lindern. Oft wird ihnen gesagt, dass ihre Symptome imaginär sind und dass nichts wirklich falsch daran ist.”
Die aktuelle Studie liefert Hinweise auf eine erhöhte TSPO-Bindung, gemessen mit CPBR28 PET, bei Patienten mit Fibromyalgie (FM) im Vergleich zu gesunden Kontrollen (HC). Dieser Marker der Gliaaktivierung war in mehreren Hirnregionen erhöht, die aus früheren Neuroimaging-Studien in die FM-Pathologie involviert waren. Wir berichten auch über positive Assoziationen zwischen dem TSPO-PET-Signal in mehreren dieser Regionen und den subjektiven Bewertungen der Müdigkeit, eines der häufigsten Symptome, über die FM-Patienten berichten (Clauw, 2014, Wolfe et al., 2011). Unsere Beobachtungen unterstützen eine Rolle für die Neuroimmun / Glia-Aktivierung in der FM-Pathologie.
Diese Ergebnisse stimmen mit einer Reihe klinischer Daten überein, die auf einen möglichen Zusammenhang zwischen Neuroinflammation und FM hindeuten. Mehrere Studien an FM-Patienten zeigten erhöhte CSF-Spiegel von Molekülen, die an neuroglialen Signalen beteiligt sind, wie z. B. Fractalkine und IL-8.
Fibromyalgie und Neuroinflammation
Entzündung ist ein zunehmend wichtiges Thema bei Fibromyalgie geworden. Es ist klar, dass bei Fibromyalgie (und anderen chronischen Schmerzstörungen) eine „zentrale Sensibilisierung“ – eine Erhöhung der vom Zentralnervensystem verursachten Schmerzempfindlichkeit – vorliegt. Entzündungsfördernde Nervenfaktoren entstehen durch einen entzündlichen Prozess, diese Nervenfaktoren versetzen die schmerzerzeugenden Nerven in Schwindel und könnten die Ursache dieser zentralen Sensibilisierung sein.
Neuroinflammation ist dann eine große Möglichkeit bei FM, aber es war nicht einfach zu messen. Erst kürzlich wurden bildgebende Verfahren für das Gehirn entwickelt, mit denen möglicherweise die bei FM vorhandene Neuroinflammation gemessen werden kann. In dieser Studie wurde versucht, die Frage der Neuroinflammation auf andere Weise zu beantworten. In letzter Zeit hat das Interesse an den verschiedenen Gehirnzellen, die bei Entzündungen und Überempfindlichkeit eine Rolle spielen, zugenommen.
Eine solche Zelle, von der bekannt ist, dass sie eine zentrale Rolle bei den Entzündungsreaktionen im Gehirn spielt, ist die Mikroglia. Das Gehirn hat eine ziemlich dichte Population von ihnen. Bevor wir uns jedoch näher mit der Rolle von Mikroglia bei Entzündungen und Hyperreaktionen des Gehirns befassen, sollten wir uns mit Entzündungen und dem Zusammenhang zwischen Erkrankungen des Gehirns und peripheren Entzündungen befassen. Schließlich wird FM häufig durch einen Gesundheitszustand ausgelöst, der periphere Organe betrifft.
Entzündungsmechanismus
Entzündung ist ein Abwehrmechanismus. Sobald der Körper einem Infektionserreger oder einem lokalen Trauma ausgesetzt ist, wird er von den als Makrophagen bezeichneten Zellen erkannt und praktisch aufgefressen (Infektionserreger, Trümmer, anderes Fremdmaterial). Makrophagen spielen auch eine zentrale Rolle bei der Initiierung einer Immunantwort, und zwar durch die Sekretion von Zytokinen und Chemokinen.
Chemokine sind die Botenstoffe, die andere Arten von Abwehrzellen in die Region locken, während Zytokine komplexere Botenstoffe sind, die Informationen über Stress und Angriffe an verschiedene Zellen und Körperteile senden. Bald nach der anfänglichen Entzündungsreaktion kommt es zu Übelkeit, was zu erhöhter Körpertemperatur, Schmerzen und einem Verlust des Interesses an allem in der Umgebung führt.
Es ist eine Zeit, in der unsere Gehirnzentren in Aktion treten. Es sind die spezifischen Zentren im Gehirn, die die Körpertemperatur erhöhen, um das mikrobielle Wachstum zu kontrollieren (Anstieg der Körpertemperatur ist ein regulierter Abwehrmechanismus). Unser Gehirn versucht, Energie zu sparen, indem es uns zwingt, eine Pause einzulegen, soziale Interaktion und körperliche Aktivitäten zu reduzieren.
Wir wissen, dass das Gehirn unabhängig vom Ort der Entzündungsreaktion eine zentrale Rolle bei der Steuerung verschiedener Reaktionen spielt. Es wirft die Frage nach dem Mechanismus auf, der dem Informationsaustausch zwischen dem Entzündungspunkt (der oft weit vom Gehirn entfernt ist) und dem Gehirn zugrunde liegt. Dies geschieht auf zwei Arten, zum einen durch die als Zytokine bezeichneten Botenstoffe und zum anderen durch die Eingaben der Nerven
Behandlung zur Senkung der Neuroinflammation
Wir haben bereits einige Beweise dafür, dass Behandlungen, die speziell das Entzündungsniveau im Gehirn senken, die Symptome der Fibromyalgie lindern können. Viele der von uns als entzündungshemmend empfundenen Medikamente (wie Ibuprofen oder andere nichtsteroidale entzündungshemmende Mittel) wirken bei Hirnentzündungen nicht gut, aber es gibt einige Behandlungen, die dies bewirken. Niedrig dosiertes Naltrexon (LDN) ist das wirksamste Medikament, das jedoch hauptsächlich von Anbietern alternativer Medizin verschrieben wird, da die meisten westlichen Ärzte für die Schmerzbehandlung nicht gut bekannt sind.
Ein opiatblockierendes Medikament ist als Naltrexon bekannt, das in höheren Dosen (50 mg) zur Behandlung von Opiat- und Alkoholabhängigkeit verschrieben wird. LDN senkt die Entzündung des Zentralnervensystems, wenn es in sehr geringen Dosen eingenommen wird (Dosierungsbereich 1–5 mg).
Es gibt spezifische Rezeptoren für die Immunzellen im Gehirn, sogenannte Gliazellen. LDN wirkt auf sie ein und fordert sie auf, in den Winterschlaf zurückzukehren und keine entzündlichen Chemikalien mehr freizusetzen. In zwei an der Stanford University durchgeführten Studien konnte gezeigt werden, dass LDN Fibromyalgie-Schmerzen spürbar lindert und auch die Schmerzüberempfindlichkeit verringert.
Kräuterbehandlung zur Behandlung von Entzündungen
Kurkuma: Dieses gelbe Gewürz wird seit Jahrhunderten als entzündungshemmendes Mittel in der ayurvedischen Medizin eingesetzt. Der aktive Bestandteil ist eine Chemikalie namens Curcumin, deren entzündungshemmende Wirkung im Gehirn nachgewiesen wurde.
Grüner Tee: Eine Chemikalie namens EGCG (-) – Epigallocatechin-3-Gallat ist nachweislich “stark abwehrend gegen Entzündungen, oxidative Schäden und Zelltod” im Gehirn. Es wird aus grünem Tee extrahiert.
Kreuzblütler: Sulforaphan ist ein Extrakt aus Brokkoli, der vor Hirnentzündungen schützt und die Schädigung von Neuronen verringert.
Zu den entzündungshemmenden Nahrungsergänzungsmitteln gehören:
- >>> Omega-3-Fettsäuren
- >>> Kurkuma >>> Ingwer
- >>> coQ10
- >>> Rhodiola rosea
Viele Ärzte empfehlen eine entzündungshemmende Diät für entzündliche Erkrankungen, aber wir haben nicht genug Forschung darüber. Menschen beginnen oft mit einer streng begrenzten Diät, weil die Entzündungsauslöser nicht bei allen gleich sind. Dann fügen sie jeweils eine Art von Lebensmittel hinzu, um zu überprüfen, welche Lebensmittel problematisch sind.
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